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Cell Metab (2018) 27(2):299313
This opens protocol design options and raises mechanistic questions about how a 15-amino-acid peptide survives the GI tract with sufficient intact molecule to produce systemic effects (or whether the observed effects are primarily local GI-tract actions)
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Inhibition of EMMPRIN and MMP-9 expression by Epigallocatechin-3-Gallate through 67-kDa laminin receptor in PMA-induced macrophages
"It is disarray of metabolism, especially when accompanied by inflammatory chemicals that can form the precursors of cancer, including colon cancer." Potential limitations The study did have some limitations, primarily that it could not prove that GLP-1s caused the extended survival, but only established an association
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